By Jean-Claude Kader, Michel Delseny
Edited by way of Jean-Claude Kader and Michel Delseny and supported via a world Editorial Board, Advances in Botanical learn publishes in-depth and up to date reports on a variety of subject matters in plant sciences. at the moment in its fiftieth quantity, the sequence includes a wide selection of stories by means of famous specialists on all elements of plant genetics, biochemistry, phone biology, molecular biology, body structure and ecology. This eclectic quantity gains six reports on state of the art issues of curiosity to postgraduates and researchers alike. * Multidisciplinary studies written from a vast variety of clinical views * For over 30 years, sequence has loved a name for excellence * participants the world over famous specialists of their respective fields
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Extra info for Advances in Botanical Research, Vol. 55
2003). The abaxial side of leaves is specified by another set of regulators, acting antagonistically to the adaxial determinants, suggesting the mutually exclusive and opposing nature of adaxial and abaxial cell fates (Fig. 2A). , 2005). , 2008). , 2005). , 1999). , 2010; Izhaki and Bowman, 2007). One important question that remains to be answered concerns how polarity information is generated to induce the expression of fate-specific regulators. It has been suggested that the establishment of the adaxial–abaxial axis of leaves is dependent on the conversion of positional signals provided by the CARPEL DEVELOPMENT 15 SAM, and probably also by other surrounding areas, into the differential expression of the mutually antagonistic transcription factors mentioned above (Fig.
Genetic pathways controlling floral meristem (FM) termination. AG is the main switch towards FM termination, but triggers different developmental programmes depending on its protein partners: when AG interacts with B-class proteins AP3 and PI, it promotes stamen development, and without B-class proteins it promotes both carpel development and FM termination. SUP, which excludes B-class gene expression from the fourth whorl, promotes these two latter functions. In whorl 4, AG activates several targets, among which KNU plays a central role in switching off WUS, and thus stem cell termination.
G. , 1995), while indeterminacy is the first phenotypic flaw associated with reduced levels of AG (Chuang and Meyerowitz, 2000; Mizukami and Ma, 1995). , 2007). However, unlike AG, none of the genes corresponding to these mutations, with the notable exception of KNU, are strictly required for the arrest of stem cell maintenance within the FM. Moreover, most of these genes appear to act upstream of AG, and thus control FM termination through this factor. , 2009). DICER-LIKE1/CARPEL FACTORY (DCL1/CAF) and HUA CARPEL DEVELOPMENT 33 SUP AP3 PI AG AG SEP SEP AG AP3 SEP PI AG KNU CRC (SPT) ?